Work
Chief engineer at the Genomics Core Facility (Next generation Sequencing) and the Genetics Group (cell culture, qPCR, nucleotide extraction, lab training of PhD candidates).
Publications
Academic article
- Bartaula-Brevik, Sushma; Leitch, Calum; Valladares, Maria del Carmen Hernandez et al. (2023). Vacuolar ATPase Is a Possible Therapeutic Target in Acute Myeloid Leukemia: Focus on Patient Heterogeneity and Treatment Toxicity. (external link)
- Nepstad, Ina; Hatfield, Kimberley Joanne; Grønningsæter, Ida Sofie et al. (2019). Effects of insulin and pathway inhibitors on the PI3K-Akt-mTOR phosphorylation profile in acute myeloid leukemia cells.. (external link)
- Brenner, Annette; Tvedt, Tor Henrik Anderson; Nepstad, Ina et al. (2017). Patients with acute myeloid leukemia can be subclassified based on the constitutive cytokine release of the leukemic cells; the possible clinical relevance and the importance of cellular iron metabolism. (external link)
- Brenner, Annette; Reikvam, Håkon; Rye, Kristin Paulsen et al. (2017). CDC25 inhibition in acute myeloid leukemia - A study of patient heterogeneity and the effects of different inhibitors. (external link)
- Kittang, Astrid Marta Olsnes; Kordasti, Shahram; Sand, Kristoffer et al. (2016). Expansion of myeloid derived suppressor cells correlates with number of T regulatory cells and disease progression in myelodysplastic syndrome. (external link)
- Bruserud, Øystein; Reikvam, Håkon; Fredly, Hanne Kristin et al. (2015). Expression of the potential therapeutic target CXXC5 in primary acute myeloid leukemia cells - high expression is associated with adverse prognosis as well as altered intracellular signaling and transcriptional regulation. (external link)
- Reikvam, Håkon; Brenner, Annette; Hagen, Karen Marie et al. (2015). The cytokine-mediated crosstalk between primary human acute myeloid cells and mesenchymal stem cells alters the local cytokine network and the global gene expression profile of the mesenchymal cells. (external link)
- Astori, Audrey; Fredly, Hanne Kristin; Aloysius, Thomas Aquinas et al. (2013). CXXC5 (retinoid-inducible nuclear factor, RINF) is a potential therapeutic target in high-risk human acute myeloid leukemia. (external link)