Overingeniør ved Kjernefasilitet for Genomikk (Dypsekvensering/Next generation sequencing) og opplæring av stipendiater og andre studenter i laboratoriet.

Vitenskapelig artikkel

• Bartaula-Brevik, Sushma; Leitch, Calum; Valladares, Maria del Carmen Hernandez et al. (2023). Vacuolar ATPase Is a Possible Therapeutic Target in Acute Myeloid Leukemia: Focus on Patient Heterogeneity and Treatment Toxicity. (ekstern lenke)
• Nepstad, Ina; Hatfield, Kimberley Joanne; Grønningsæter, Ida Sofie et al. (2019). Effects of insulin and pathway inhibitors on the PI3K-Akt-mTOR phosphorylation profile in acute myeloid leukemia cells.. (ekstern lenke)
• Brenner, Annette; Reikvam, Håkon; Rye, Kristin Paulsen et al. (2017). CDC25 inhibition in acute myeloid leukemia - A study of patient heterogeneity and the effects of different inhibitors. (ekstern lenke)
• Brenner, Annette; Tvedt, Tor Henrik Anderson; Nepstad, Ina et al. (2017). Patients with acute myeloid leukemia can be subclassified based on the constitutive cytokine release of the leukemic cells; the possible clinical relevance and the importance of cellular iron metabolism. (ekstern lenke)
• Kittang, Astrid Marta Olsnes; Kordasti, Shahram; Sand, Kristoffer et al. (2016). Expansion of myeloid derived suppressor cells correlates with number of T regulatory cells and disease progression in myelodysplastic syndrome. (ekstern lenke)
• Reikvam, Håkon; Brenner, Annette; Hagen, Karen Marie et al. (2015). The cytokine-mediated crosstalk between primary human acute myeloid cells and mesenchymal stem cells alters the local cytokine network and the global gene expression profile of the mesenchymal cells. (ekstern lenke)
• Bruserud, Øystein; Reikvam, Håkon; Fredly, Hanne Kristin et al. (2015). Expression of the potential therapeutic target CXXC5 in primary acute myeloid leukemia cells - high expression is associated with adverse prognosis as well as altered intracellular signaling and transcriptional regulation. (ekstern lenke)
• Astori, Audrey; Fredly, Hanne Kristin; Aloysius, Thomas Aquinas et al. (2013). CXXC5 (retinoid-inducible nuclear factor, RINF) is a potential therapeutic target in high-risk human acute myeloid leukemia. (ekstern lenke)

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